Lipid peroxidation in the etiopathogenesis of inflammatory periodontal disease
Abstract
Reactive oxygen species are involved in the etiopathogenesis of inflammation. During the activation of leukocytes, large quantities of these species are released,whose function is the elimination of pathogens. If the antioxidant defenses of the tissues do not work efficiently, radical reactions are induced that affect the
biomolecules. The attack to the lipids of the cellular membrane causes its peroxidation, with the consequent formation of new radical species and toxic metabolites. The presence of a strong
Inflammatory infiltrate in periodontal tissues, during inflammatory periodontal disease, has suggested the possible participation of reactive oxygen species in the
etiopathogenesis of this disease. It is assumed that the attack of these substances on periodontal tissues with deficient antioxidant defense, causes the appearance of lipid peroxidation,
which can lead to cell lysis and protease activation.
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